A complete history is necessary to determine the severity of
illness and to identify the type of dehydration present.
Obtain as much information from the child, and elicit
further details and clarifications from the parent or care
giver. Obtain a detailed description of intake (types of
liquids and solids, volume, frequency) and output of urine
(frequency, amount, color, odor, hematuria), stool
(number, consistency, presence of blood or mucous), and
emesis (frequency, volume, bilious or nonbilious,
hematemesis). Estimate urine output by the number and
saturation of wet diapers in infants and young children.
Note the presence of abdominal pain (duration, location,
intensity, quality, and radiation). Inquire about weight loss
and activity level. Note the time interval of symptoms. The
last episode of vomiting is important in determining when
the initiation of an oral trial is advisable.
Ask about associated symptoms (fever, headache, neck
pain, throat pain, dysuria, urinary frequency, rash). Travel
and recent antibiotic use are also pertinent.
should be considered. Important elements of the past medical
history include immunocompromise and malignancy.
The examination begins with assessment of the general
appearance of the child. Lethargy or listlessness can warn
of impending circulatory collapse. Examine the throat for
erythema, ulcerations, or tonsillar exudates. Assess the
abdomen for tenderness, rebound, or guarding.
Neurologic exam should include mental status, cranial
nerves, strength, and reflexes. Altered mental status or
focal neurologic findings can indicate increased intracra
nial pressure. Capillary refill and skin turgor should be
noted. The gold standard for the diagnosis of dehydration
is measurement of acute weight loss. True pre-illness
weight is rarely known in the acute care setting. An
estimate of the fluid deficit is thus made based on clinical
assessment ( Table 5 1 -1). Any of the two following findings
branes, and delayed capillary refill ( > 2 seconds). Other
important considerations are abnormal respiratory
Vital signs are an important objective measure and can
be normal in a child with dehydration. The first sign of
mild dehydration in children is tachycardia. Hypotension
is a late sign of severe dehydration.
altered mental status. Blood sugar may be low (poor
point to a specific diagnosis: high K (congenital adrenal
hyperplasia, renal failure), low K (pyloric stenosis), low
bicarbonate (acidosis, HC03 loss in diarrhea), high blood
urea nitrogen/creatinine (renal hypoperfusion). Urinalysis
may show glucose, ketones, or signs of infection. Urine
specific gravity may be elevated in patients with dehydra
tion, but it is not a reliable measure. Serum sodium should
be determined because hypo/hypernatrernia requires spe
No imaging is required for most patients presenting to the
ED with dehydration. Consider flat/upright abdominal
x-rays if there is suspicion for obstruction. Ultrasound or
computed tomography ( CT) scan of the pelvis is indicated
if appendicitis suspected. Noncontrast head CT scan is
indicated when evaluating severe headache or if exam
reveals signs of intracranial pressure.
Table 51-1. Cli nical assessment of severity of dehydration in the pediatric patient.
Mental status Alert/restless Irritable and drowsy Lethargic
Respirations Normal Deep ± rapid Deep and rapid
Pulse Normal Rapid and weak Weak to absent
Blood pressure Normal Normal with orthostasis Low
Mucous membranes Moist Dry Very dry
Tears Present Decreased Absent
Skin turgor Pinch and retract Tenting Tenting to doughy
Urine output Normal Decreased Absent
Capillary refill <2 sec 2-3 sec >3 sec
History and physical examination are generally sufficient
to identify signs or symptoms of dehydration. Shock needs
The underlying cause of dehydration should be identified,
Identify patients with signs of shock and resuscitate with
fluid immediately (20 mL/kg normal saline [NS] or
Lactated Ringer's over a 20 - to 30-minute period). Reassess
and repeat fluid bolus until perfusion is adequate and vital
signs normalize (fluid bolus x 3 if necessary). Urine output
is the most important indicator of restored intravascular
causes of shock (sepsis, hemorrhage, cardiac disease). Treat
hypoglycemia promptly (2.5 mL/kg of 10o/o dextrose or
1 mL/kg of 25o/o dextrose). Once vital sign abnormalities
are corrected, the rate of fluid administration for treatment
is determined by the estimated fluid losses plus ongoing
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