Search This Blog

468x60.

728x90


(ii) Zoophilic species: These are the dermatophytes that live on animals and often cause infection in

their animal host. These zoophilic species are transmitted from infected animals to humans by direct

and indirect contacts with domestic animals (e.g., cat and dog) and occasionally wild animals.

Examples are Trichophyton violaceum and Microsporum canis.

(iii) Geophilic species: These are saprophytic fungi found in

soil or in dead organic substances. They occasionally cause infection in humans and animals.

Examples are Microsporum gypseum and Trichophyton ajelloi. Dermatophytes usually grow only on

keratinized skin and do not penetrate the living tissues. In some infected persons, hypersensitivity to

fungus antigen may cause secondary

eruptions, such as vesicles on the finger. This reaction is known as dermatophytid (id) reaction. This

reaction occurs as a result of hypersensitivity response to circulating fungal antigen, and these

lesions do not contain any fungal hyphae.

Laboratory diagnosis

Laboratory diagnosis is based on demonstration of fungal element in clinical specimen by

microscopy and confirmation by culture. The specimens include skin scrapings and nail clippings or

hair taken from the areas suspected to be infected by dermatophytes. These entire specimens are

treated with alkali solution to clear epithelial cells and other debris. Direct microscopy is useful only

for diagnosis, while culture is always carried out to identify the specific causative fungal agent.

Direct microscopy

Examination of 10% direct KOH mount may show fungal

hyphae. Three types of hair infections can be demonstrated in

microscopy of 10% KOH wet mount as follows (Fig. 4-1):

Ectothrix: Ectothrix infection is characterized by presence of a layer of arthrospores on the surface of

hair shaft . It is caused by M. audouinii, M. canis, and Trichophyton mentagrophytes.

501

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

Endothrix: The clusters of arthrospores are found entirely

within the hair shaft in endothrix infection . It is caused by Trichophyton tonsurans, T. violaceum, and

Trichophyton schoenleinii.

Culture

The clinical specimens are cultured by inoculation on SDA containing antibiotics like cycloheximide.

The media after inoculation are incubated at 25–30°C for 3 weeks. At 25°C most of the pathogenic

fungi grow well, while saprophytic fungi and bacteria are inhibited.

The cultures are examined at regular intervals, and dermatophytes

are identified based on (a) colony morphology, ( b) pigment

production, and (c) presence of microconidia and macroconidia.


Subcutaneous Mycosis

Subcutaneous mycosis is defined as fungal infection associated

with development of characteristic lesion in subcutaneous tissue and overlying skin with or without

extension to bone and muscle. This is caused by a heterogeneous group of fungal infection of low

pathogenic potential introduced in the body percutaneously from a trivial trauma. shows the

classification of subcutaneous mycoses.

Mycetoma

Mycetoma is a slowly progressive, chronic granulomatous infection of skin and subcutaneous tissues

with occasional involvement of underlying fascia and bone usually affecting

extremities.

Figure (4-1) Endothrix and Ectothrix infection

502

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

extremities are most commonly involved. Microabscesses are formed in subcutaneous tissues

surrounded by polymorphonuclear inflammatory reaction. The center of the lesion consists of

tangled filaments of these organisms.

During the course of infection, microabscesses burst open

with the formation of chronic multiple sinuses discharging copious, seropurulent fluid containing

granules. The color and consistency of these granules vary depending on the fungi that cause the

disease.The condition is characterized by formation of painless, localized, swollen lesions on the

affected limbs.Multiple discharging sinuses are present.

Systemic mycoses

Systemic mycoses are caused by fungi of soil, which are inherently virulent and cause disease in

healthy humans. The systemic ycoses include coccidioidomycosis, paracoccidioidomycosis,

histoplasmosis, blastomycosis, and cryptococcosis.

Histoplasmosis

Histoplasmosis is primarily a disease of reticuloendothelial system caused by an intracellular fungus

Histoplasma capsulatum. H. capsulatum is a dimorphic fungus, which occurs in two stages: as a mold in

soil and as yeast at body temperature in mammals. On SDA medium at 37°C, this fungus produces

cottony mycelial growth. The colony is characterized by thin, branching, septate hyphae that produce

tuberculate macroconidia and microconidia.

Blastomycosis

Blastomycosis is a granulomatous fungal infection caused by B. dermatitidis. B. dermatitidis is a

dimorphic fungus, which occurs in two stages: as mold in soil and as yeast in tissue.

On culture at 37°C and in tissue, the yeast is a round structure

with a double refractile wall and a single broad-based bud. This appearance helps to differentiate it

from the Cryptococcus neoformans yeast, which has a narrow-based bud. On culture at 25°C, the

fungus produces a mycelial growth showing typical pyriform microconidia, which measure 2–4 _m

in diameter.

Cryptococcosis

Cryptococcosis, also called European blastomycosis, is an acute to chronic disease caused by an

encapsulated yeast, C. neoformans.

Cryptococcosis is the most common life-threatening fungal disease in patients with AIDS Of the 19

species that comprise the genus Cryptococcus, human disease is associated with only C. neoformans.

503

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

Morphology

 C. neoformans is a true yeast.

 It is an oval and budding cyst and measures 3–6 _m in diameter. The yeast may be single or

may have a single budding daughter cell.

 Within the host and in certain culture media, the yeast is urrounded by a wide

polysaccharide capsule. The polysaccharide capsule is composed of mannose, xylose, and

glucuronic acid.

 C. neoformans on SDA medium forms smooth, convex, cream-colored colonies at 20–37°C.

Lactophenol cotton blue (LPCB) wet mount of the colony shows budding yeast cells.

C. neoformans has two varieties: C. neoformans var neoformans and C. neoformans var gattii. Based on

antigenic specificity of the capsular polysaccharide, the species has been classified into four

serotypes. These are serotypes A and D (C. neoformans var neoformans) and serotypes B and C (C.

neoformans var gattii).

Pathogenesis and Immunity

The immune status of the host is the crucial factor in pathogenesis

of cryptococcosis. C. neoformans usually causes most serious infections in patients with

impaired CMI. These include:

 patients with AIDS,

 patients undergoing corticosteroid treatment,

 patients undergoing organ transplantation,

 patients with reticuloendothelial malignancy, and

 patients with sarcoidosis.

C. neoformans is primarily transmitted by inhalation . Following inhalation, the yeasts are deposited

into the pulmonary alveoli, in which they survive before they are phagocytosed by alveolar

macrophages. Glucosylceramide synthase has been identified as an essential factor in the survival of

C. neoformans in pulmonary alveoli.

Cryptococcal polysaccharide capsule has antiphagocytic properties. Hence, unencapsulated yeast are

readily phagocytosed and destroyed than the encapsulated organisms, which are more resistant to

phagocytosis. The antiphagocytic properties of the capsule prevent recognition of the yeast by

phagocytes and inhibit leukocyte migration into the area of fungal replication.

Host immunity

The host immunity in cryptococcal infection is mediated by both cellular and humoral responses.

CMI is mediated by natural killer cells and T lymphocytes can inhibit or kill cryptococci. An increase

in helper T-cell activity, skin test conversion, and a reduction in the number of viable organisms in

the tissues indicates a successful host response against the fungus. Humoral immunity is mediated

504

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

by anticryptococcal antibodies and soluble anticryptococcal factors. Both anticryptococcal antibodies

and the complement play a crucial

role in facilitating the macrophage- and lymphocyte-mediated immune response to the organism.

Clinical Syndromes

C.neoformans causes (a) pulmonary cryptococcosis in immunocompetent hosts and in

immunocompromised hosts, (b) CNS

cryptococcosis, and (c) disseminated nonpulmonary non-CNS cryptococcosis.

o Pulmonary cryptococcosis

The clinical manifestations of pulmonary cryptococcosis are widely variable. Pulmonary disease

varies from asymptomatic colonization of the respiratory tract to acute respiratory distress syndrome

ffecting immunocompromised hosts. It depends on the immune status of the host

o CNS cryptococcosis

Both the brain and meninges are involved in cryptococcal infection of the CNS infections. Meningitis

and meningoencephalitis are the most common manifestations. These are usually subacute or chronic

in nature. Without specific therapy, the infection is invariably fatal. The patient dies due to the

disease 2 weeks to several years after the symptom onset.

o Disseminated nonpulmonary non-CNS cryptococcosis

Disseminated cryptococcosis includes the skin, prostate, and medullary cavity of the bones, next only

to the lungs and CNS, and it occurs most commonly in patients with AIDS and other

immunosuppressed conditions.

Epidemiology

C. neoformans is distributed worldwide. The incidence of cryptococcosis is increasing and now it represents a

major lifethreatening fungal infection in patients with AIDS. Most cases of cryptococcosis are caused by

serotypes A and D. C. neoformans var gattii is the most common variety that causes disease in

immunocompetent patients. C. neoformans var neoformans is the most common variety that causes disease in

immunocompromised

patients, e.g., AIDS. C.neoformans is primarily transmitted by inhalation

(Figure 4-2). Human-to-human transmission does not occur.

505

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

Laboratory Diagnosis

Laboratory diagnosis of cryptococcal infection is made by demonstration of the yeast in CSF,

sputum, pus, and brain biopsy tissue by smear and culture. Methenamine silver or periodic acidSchiff stains are used to stain the tissue specimens for demonstration

of the capsule of C. neoformans. Fixed tissue may also be stained

with mucicarmine, which preferentially stains C. neoformans.

India ink preparation is commonly used to detect budding yeast cells in the CSF (Figure 4-3). The

capsule appears as a clear halo around the yeast cells. By this method, cryptococci can be

demonstrated in 25–50% of patients with cryptococcal meningitis. Gram-stained smear of the CSF

shows Gram-positive yeast cells . The culture of centrifuged CSF specimens confirms diagnosis of the

condition. This fungus is identified based on the macroscopically appearance ,biochemical test result

and ability to grow at 37 C° .

Figure (4-2) Transmission of Cryptococcus neoformans

506

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

Opportunistic Fungal infections

The opportunistic fungi usually cause infections in persons with impaired host defense, but do not

cause disease in most of the immunocompetent hosts. Since these fungi become pathogens in

individuals with impaired immunity by taking

advantage of the host’s debilitated conditions, they are called opportunistic fungi.

The most opportunistic infections are caused by Candida albicans, Aspergillus spp., Penicillium marneffei,

and various Zygomycetes (Table 4-4).

Candidiasis

Candida species are the most common fungal pathogens that affect humans. These species are true

opportunistic pathogens that take advantage of the host’s debilitated condition and gain access to the

circulation and deep tissues. The genus Candida includes more than 100 species, of which only few

cause disease in humans. C. albicans and occasionally other species cause candidiasis, a major

infection in immunocompromised .

Figure(4-3) India ink preparation showing capsule of

Cryptococcus neoformans (_400).

507

Arranged by Sarah Mohssen

Section IV - Medical Mycology By Dr. Kareem Lilo

Table (4-4 ) Common opportunistic infections

Candida albicans

C. albicans is the most common Candida species, which causes

opportunistic infections in immunocompromised hosts. It

forms the part of the normal flora of the mucous membrane of

the gastrointestinal, genitourinary, and respiratory tract.

No comments:

Post a Comment

اكتب تعليق حول الموضوع

mcq general

 

Search This Blog